Obesity and heart: Cardiologists view | 48576

Primary Health Care: Open Access

ISSN - 2167-1079

Obesity and heart: Cardiologists view

Annual Congress & Medicare Expo on Primary Healthcare

April 25-27, 2016 Dubai, UAE

Schusterova I and Tohatyova A

P J S University in Kosice, Slovakia

Scientific Tracks Abstracts: Primary Health Care

Abstract :

Obesity causes chronic volume overload (increased preload) and dilation of the left ventricle (LV) of the heart. Hypertrophy LV itself as an adaptation to expanded intravascular volume determines the damage of the diastolic function LV especially by influencing later diastolic passive infilling. For persistent obesity they may of course result in systolic dysfunction and manifest heart failure while damaging the function of the myocardium correlates with body mass index and the duration of obesity. The clinical picture of the consequences of cardiomyopathy depends on the severity of the emerging cardiac changes, age, duration and severity of the obesity, as well as on associated diseases, which often accompany obesity (arterial hypertension, disorder in the metabolism of lipids and glucose, ischemic heart disease, sleep apnea syndrome, etc.). Studies of obese child patients indicate, initial signs of cardiomyopathy ΓΆΒ?Β? changes of indices of diastolic and systolic functions, though they are clinically silent. Early structural and functional changes in the myocardium of obese children and overweight children represent a higher risk of clinical manifestation of this cardiomyopathy in early adulthood, and therefore they require increased vigilance of the pediatricians and pediatric cardiologists. In one of our study, we confirmed differences not only in LVM (p<0.001) and LVM index body height2.7 (p<0.05) but as well as the differences in left ventricle size (IVS (p<0.01), LVEDD (p<0.005), LVESD (p<0.05), PWTh (p<0.001)) and volumes include stroke volume (p<0.05) in spite of normal preserved systolic function in obese and overweight children. Increased LV mass in obese individuals is also a result of the increased amount of epicardial fat and fat infiltration of the myocardium. The thickness of epicardial adipose tissue (EAT) as measured by echocardiography represents an independent predictor of visceral adiposity. A very good positive correlation was found between echocardiographic measurements of EAT and MRI measurements, which represent the gold standard for assessing visceral fat mass. We confirmed significantly higher thickness of echocardiography EAT was found in overweight and obese adults and children (p<0.001). However, it requires the creation of a standardized examination methodology for children age, as well as the creation of certain reference values while considering the growth and development of children.

Biography :

Schusterová I is an Associate Professor and the Head of the Department of Paediatrics and Adolescent Medicine, Children University Hospital, P J Š University in Košice, Slovakia. At the same time, she is the Head Physician in the Tertiary Center for Valvular and Congenital Heart Defects, Eastern Slovak Institute of Cardiac and Vascular Diseases. She took specialization exam from Adult and Pediatric Cardiology at National Institute of Cardiac and Vascular Diseases in Bratislava, Slovak Republic. After Graduation, she took part in Fellowship Training at Ottawa Civic Hospital, and later on performed research work in the area of Cardiology at University of Ottawa in Canada. In the period from 11/1999 till 7/2004, she completed her training at Cardiologic Department, II. Internal Medicine Clinic, General Hospital, Vienna under supervision of Professor Dr Gerald Maurer. Later in 2005, she broadened her professional knowledge and skills at Cardiac Surgery Clinic, Kardiocentrum Universitätsklinik in Bad Oyenhausen, Germany. Her research interests focus on preventive cardiology, obesity, and disorders of lipid metabolism, atherosclerosis and their prevention in the paediatric age group.