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The psychotherapeutic considerations: brain behavior against | 84319

Neurology and Neurorehabilitation

Abstract

The psychotherapeutic considerations: brain behavior against grief as psychopharmacological support in adulthood, is a research proposal that involves scientific methods within the psychotherapeutic dispositions that currently are developed in front of patients who experience pathologies related to the different experiences that from the duel can develop a subject; this is how the present work tries to determine the organization of individualized psychological and psychopharmacological therapy (Psychiatry) based on the results that exposes the brain activity of a subject and sustained by the neuronal disconnections suffered by an individual suffering a stage of grief, developing pathologies such as: depression, anxiety, stress and even reach suicide. Similarly, it is intended to demonstrate and raise awareness that the changing brain activity of those who suffer a grief should be treated psychopharmacologically distinct, individualized and with clinical supports that contribute to improving the quality of life that a patient experiences during this stage.

Malcolm R Hooper

Almost 20 to 30% of the body’s consumption of oxygen occurs within 3 to 5% of the body mass; the brain and spinal cord structures. These structures are extremely sensitive to oxygen deficiency. The final frontier in the treatment of degenerative neurovascular disorders including brain and spinal cord injury is focused on repair and functional restoration. This involves the use of neural growth factors to promote axonal sprouting, activation of idling and nonfunctional neurons whilst promoting neovascularization (new capillary formation) of the damaged (Penumbra) areas. The extent of neurovascular deterioration can be significantly diminished with Hyperbaric Oxygenation (HBOT), which expands the therapeutic window. HBOT primes the body and provides a fertile neurovascular platform for mobilizing the patient’s own immune and circulating stem cell capacity. HBOT activates dormant and inactive nerve cells and promotes plasticity to hasten recovery. A single two hour exposure to HBOT at 2 ATA doubles circulating CD34+ progenitor stem cells (primordial cells targeted to salvage and restore damaged structures) and at approximately 40 hours HBOT, stem cell activity increases eight fold (800%)..

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